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After the knockout of the GLP-1R gene, in transgenic mice expressing human GLP-1R in pancreatic islets and pancreatic ductal cells restored cAMP and serine threonine kinase (Akt) phosphorylation levels in isolated islets, increased GLP-1-stimulated insulin secretion and GLP-1R-dependent -cell proliferation, and promoted transgenic mice for glucose regulation in response to feeding (Lamont et al., 2012)

Disruption of the nonneuronal tph1 gene demonstrates the importance of peripheral serotonin in cardiac function

We first tested the effect of the antagonists either alone or in combination in DIO mice due to their potential effects by blocking endogenous glucagon or GLP-1

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